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People with autism have significantly more synapses than healthy people

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People with autism have significantly more synapses than healthy people

PantherMedia

Dangerous child's play

Autism is a neurodevelopmental disorder and its first symptoms appear in early childhood. Importantly, they last a lifetime. We know of two types of autism (schizophrenic and infantile). The cause of the development of this disease is not yet known, although we know that both biological and psychological factors have a huge impact on its development. Scientific studies, show that people with autism have many more synapses in their brain than healthy people, as they do not undergo a natural pruning process.

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Autism is a disease that causes the patient to 'cut off, detach' from the world around them, have a very limited exchange of information with the environment and present a subjective isolation. One factor that has a huge impact on the development of autism is brain damage. These occur as a result of the appearance of teratogens during pregnancy and birth. Micro-damage to the brain also occurs as a result of hypoxia, arising in the fetal or neonatal period. Their consequence is any disturbance or impairment in the patient's emotional and personality spheres.

A scientific study conducted by scientists at USC (Columbia University) and published in a scientific journal, shows that autistic patients have too many synaptic connections in their brain. Depending on the type of cells between which the signal is transmitted, there are three types of such connections: neural-nerve, neuromuscular and neuromuscular.

Too many synapses can be a major cause of abnormal brain function. Scientific studies conducted on animal material (mice), show that stimulating the process of 'pruning' syn apses leads to improvements in the functioning of these animals and, importantly, eliminates symptoms of autism. The pruning of synapses is a process that is natural and even necessary. It occurs in the brain in every healthy person during their adolescence.

Research is still ongoing, with the primary aim of answering the question: why does the natural pruning of synapses not occur in autistic people? To what extent is overactivity of the mtTOR kinase responsible for this?